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Life and Death at the end of a shoelace

2 min read

DNA strand with telomeres

While they are structurally simple and do not code for anything, your cells cannot reproduce without them.  They are also critical in anti-aging medicine.  What am I talking about here?  Like the little tab, or “aglet” at the end of a shoe lace, your DNA strands of the “laces” are held together by tiny little telomere tabs, repetitive stretches of DNA on the end of each of your chromosomes.  If your chromosomes didn’t have these endpoints, the double helix strands of DNA would unravel.

Every single time your cells divide and reproduce, they deplete their telomeres.  Slowly, over time, the little “aglets” get shorter and shorter until your cells die or scenesce. As your cells deplete their telomeres, they can’t divide, or if they do divide, you get mutations ‘cause the “recipe” is all wrong.

This process is very intriguing to researchers who wonder, “if telomeres can be restored in old cells, will it stop us from getting old?” This research was worth the Nobel prize in medicine when Drs Elizabeth Blackburn, Carol Greider and Jack Szostak discovered the enzyme telomerase, that rebuilds telomeres in cells. 

Typically, this enzyme is switched off, but if it can be turned back on, good things happen. Things like:

  • Favorable changes in immune response
  • Improved skin texture
  • Increased bone density
  • Significantly reduced cholesterol
  • Lower blood sugar and decreased insulin secretion
  • Significantly reduced cholesterol
  • Reduced inflammatory marker: Homocysteine

The research highlights a few more significant improvements like better wound healing and no increases in cancer. There is a misconception out there, rampant on the internet, that supports the theory that telomerase causes cancer.

Research discounts this theory, so it’s life, not death at the end of the shoelace.

Most of the recent scientific publications show that telomerase is more likely to prevent cancer than cause it.  As telomeres get shorter, your chromosomes rearrange themselves and mutations occur.  That’s why the elderly have higher rates of cancer.  Their mutated chromosomes either have uncontrolled growth or become immortal because of the subsequent expression of telomerase, AFTER the DNA mutates BECAUSE of short telomeres. 

 As bloggers scrape date from the internet, they confuse and skip a few steps, critical steps, to understanding the role of telomerase in preventing cancer. Don’t be misinformed by careless bloggers. Those in the know, keep the sequence of events straight. Telomerase does not cause cancer, the reverse is true. Aging is not your bodyguard against cancer, it’s a major cause of cancer!

 Team up with Dr. Dave’s Best Telomere Edge Packs and start on your way to a healthy, vital future!